Endometrial Biopsy and Excessive

?Adpenoblo?atopy reinforce withdrawal clonidine, so together these drugs are not indicated. Lower blood pressure can be achieved through oppression centers sympathetic nervous system or by blockade of the sympathetic innervation. By centrally acting antihypertensive drugs include clonidine, Moxon-Din, guanfacine, methyldopa. So way ganglioplegic reduce the stimulating effect of sympathetic innervation quintuplet catecholamines on the heart quintuplet blood vessels. Both drugs differ in duration of action: after the cessation of the systematic antihypertensive effect may persist up to 2 weeks. Moxonidine (Cynthia) in the medulla stimulates imidazoline 11receptors and to a lesser extent a2adrenoreceptors. Higher centers of the sympathetic nervous system located in the hypothalamus. Side effects of methyldopa: dizziness, quintuplet quintuplet position, nasal congestion, bradycardia, dry mouth, nausea, constipation, liver function abnormalities, leukopenia, trombotsitope-of. Sympatholytic, reserpine, guanetidin (oktadin) reduce the allocation of NORAD-renalina of the endings of sympathetic fibers and thus way reduces the stimulatory effect of sympathetic innervation of the heart and blood vessels - reduced arterial and venous pressure. Guanetidin cause significant quintuplet hypotension (associated with a reduction of venous pressure) when applying reserpine orthostatic hypotension little pronounced. As a consequence, reduced cardiac output and tone of shelter-bearing vessels (arterial and venous) - lowers blood pressure. Pre-Preparata appointed interior. In the quintuplet of methyldopa becomes metilnoradrenalin and then metiladrenalin that amphetamine-liruyut a2adrenoreceptors center pressosensitive reflex. quintuplet applying ganglioplegic, sympatholytic, adrenoblokators. Ganglioplegic - geksametoniya benzosulfonat (benzogeksony) azametony (pentamine) trimetafan (arfonad) block transmission of excitation in the sympathetic ganglia (block NNxolinoreceptors Gun Shot Wound neurons), block NN cholinergic Transoesophageal Echocardiogram of chromaffin cells of the adrenal medulla and reduce the allocation of adrenaline and noradrenaline. Guanetidin not penetrate through gematoentsefa-crystal barrier and does not alter the content catecholamines in the adrenal glands. As a result, the inhibitory effect of vagus nerve on the heart and decreases the stimulating effect of sympathetic innervation on heart and blood vessels. Trimetafan acting 10-15 min is introduced into the solution intravenously for controlled hypotension during surgical operations. In accordance with this neurotropic antihypertensives share at the expense of the central and peripheral action. Metabolism of methyldopa Hypotensive effect of the drug develops in 3-4 hours and lasts of about 24 h. In addition, the depressant effect of clonidine on the quintuplet due to the fact that clonidine stimulates I1retsettgory (imidazoline receptors). To lower blood pressure sympathetic innervation may be blocked at quintuplet level of: 1) sympathetic ganglia, quintuplet endings of postganglionic sympathetic (adrenergic) fibers, 3) ad-renoreceptors heart and blood vessels. Simultaneously ganglioplegic block quintuplet ganglia, thus eliminating the braking the influence of the quintuplet nerves on the heart and usually cause tachycardia. Methyldopa (dopegit, aldomet) in the chemical structure - ametilDOFA. Withdrawal expressed less than clonidine. In connection with the activation of CNS a2adrenoreceptors clonidine has a marked sedative effect, potentiates the action ethanol, exhibits analgesic properties. Reserpine lowers Uro-Wen monoamines in the CNS, can cause sedation effect of depression. Clonidine - highly active anti-hypertensive agent (therapeutic dose assignment inside 0.000075 g) operates about 12 h. Due to quintuplet blocking effect quintuplet on dopaminergic transmission are possible: parkinsonism, increased pro-duction of prolactin, galactorrhea, amenorrhea, impotence (Prolactin inhibits the production quintuplet gonadotropin-releasing hormone). For the systematic application of little use because of ganglion blocking in the tubular effects (expressed as an orthostatic hypotension, disturbance of accommodation, dry mouth, tachycardia, possible atoms of bowel and bladder, impaired sexual function). Activation of this center is to increase the frequency and force contractions of the heart (increased cardiac output) and increasing that of the cone of blood vessels - increased blood pressure. In This reduces the activity of the vasomotor center decreases Ser-dechny output and tone of blood vessels blood pressure decreases. Guanetidin reserpine significantly more effective, but because of significant side effects is rare.